《骨骼肌》是該領域唯一一本開放獲取的期刊，出版新穎、前沿的研究和技術進步，研究骨骼肌生物學的分子機制。該雜志歡迎有關骨骼肌發育、代謝、質量和功能調節、衰老、退化、營養不良和再生等方面的手稿，反映了這一領域的廣泛研究，重點是了解成年骨骼肌、其維持及其與非肌肉細胞類型和調節調節劑的相互作用。主要關注的領域包括:骨骼肌分化骨骼肌萎縮和肥大骨骼肌老化骨骼肌的再生和退化衛星和類衛星細胞的生物學骨骼肌營養不良變性骨骼肌能量和葡萄糖穩態骨骼肌的非營養不良遺傳病，如脊髓性肌肉萎縮和肌病維持神經肌肉連接阿諾定受體的作用和鈣信號在骨骼肌核受體在骨骼肌中的作用GPCRs和GPCR信號在骨骼肌中的作用骨骼肌生物學的其他相關方面此外，還將發表關于骨骼肌分子和細胞機制的轉化臨床研究的文章。也鼓勵提交病例報告。骨骼肌反映了對骨骼肌研究的廣度，并在心臟細胞生物學和神經生物學等不同科學領域之間架起了橋梁，這些領域在細胞分化、興奮膜、細胞間通訊和維持方面具有共同的特征。合適的文章是模型和機制驅動的，并適用于適當的統計原則;純描述性研究的興趣較小。

The only open access journal in its field, Skeletal Muscle publishes novel, cutting-edge research and technological advancements that investigate the molecular mechanisms underlying the biology of skeletal muscle. Reflecting the breadth of research in this area, the journal welcomes manuscripts about the development, metabolism, the regulation of mass and function, aging, degeneration, dystrophy and regeneration of skeletal muscle, with an emphasis on understanding adult skeletal muscle, its maintenance, and its interactions with non-muscle cell types and regulatory modulators.Main areas of interest include:differentiation of skeletal muscleatrophy and hypertrophy of skeletal muscleaging of skeletal muscleregeneration and degeneration of skeletal musclebiology of satellite and satellite-like cellsdystrophic degeneration of skeletal muscleenergy and glucose homeostasis in skeletal musclenon-dystrophic genetic diseases of skeletal muscle, such as Spinal Muscular Atrophy and myopathiesmaintenance of neuromuscular junctionsroles of ryanodine receptors and calcium signaling in skeletal muscleroles of nuclear receptors in skeletal muscleroles of GPCRs and GPCR signaling in skeletal muscleother relevant aspects of skeletal muscle biologyIn addition, articles on translational clinical studies that address molecular and cellular mechanisms of skeletal muscle will be published. Case reports are also encouraged for submission.Skeletal Muscle reflects the breadth of research on skeletal muscle and bridges gaps between diverse areas of science for example cardiac cell biology and neurobiology, which share common features with respect to cell differentiation, excitatory membranes, cell-cell communication, and maintenance. Suitable articles are model and mechanism-driven, and apply statistical principles where appropriate; purely descriptive studies are of lesser interest.